The guide below is about orthostatic Hypotension (OH), nursing diagnosis for Orthostatic Hypotension, and nursing treatments for Orthostatic Hypotension. It can be used to create educational nursing care plans for OH.
Normally when changing position from lying to standing, systolic blood pressure remains unchanged or drops slightly, while diastolic blood pressure rises slightly (Hogan-Quigley et al., 2012). However, sometimes abnormal drops in pressure occur with positional changes.
The pathophysiological cause for positional drops in blood pressure is related to the dependent pooling of blood in the veins of the liver, intestines, organs, legs, and feet. Venous return and stroke volume decrease as a result of the pooled blood, so cardiac output becomes compromised and arterial blood pressure drops (Hogan-Quigley et al., 2012; Mosnaim et al., 2010). Gravity also causes blood to rush downward from the torso to the feet upon standing, further decreasing venous return (Karch, 2006).
An estimated 500-1,000 mL of blood pools in the lower extremities and splanchnic circulation upon standing (Lanier et al., 2011). Finally, hypertensive patients may additionally experience transient cerebral ischemia from decreased cerebral perfusion during acute orthostatic drops in pressure (Gangavati et al., 2011), resulting in the neurologic symptoms of OH (Lanier et al.).
Once venous return and stroke volume are compromised during orthostasis, the body attempts to compensate by activating baroreceptors located in the aorta and carotid arteries (Karch, 2006). Baroreceptors are stimulated by pressure exerted upon them by blood in the vessels. When blood pools in lower extremities, upper body vessel pressure falls, baroreceptor stimulation stops, and this information is transmitted to the brain.
The medulla then reacts by initiating vasoconstriction, increasing heart rate and cardiac output to return pressure to vessel walls and raise blood pressure. Baroreceptors work constantly to maintain a normal range of blood pressure (Karch). With inadequate response by this autonomic system, OH becomes an issue (Mosnaim et al., 2010).
The compensatory work of the baroreceptors is not immediate so it may take several minutes to restore normal blood pressure after a drop. For this reason many resources define OH as a drop in pressure within 3 minutes of standing (Hogan-Quigley et al., 2012; Lanier et al., 2011; Mosnaim et al., 2010). However, a recent study suggests that orthostatic drops in blood pressure may occur much earlier than 3 minutes, and that earlier blood pressure measurement (such as after 1 minute) may be more accurate in detecting OH and in linking OH to falls (Gangavati et al., 2011).
Many factors can affect blood pressure in patients with OH. As people age, the heart’s pumping action loses efficiency and blood vessels stiffen and weaken, causing decreased cardiac output, which contributes to decreased compensation after orthostasis. In addition, baroreceptors become less sensitive to pressure changes and the signals they send to the medulla are not as effective with age. As a result, blood pressure may drop significantly without immediate compensation (Mosnaim et al., 2010). Finally, some patients experience the phenomenon of postprandial hypotension, whereby systolic blood pressure may drop by 20 points within 75 minutes of a large meal, especially if the meal includes high levels of carbohydrates (Lanier et al., 2011).
There is also evidence to support a connection between hypertension (HTN) and OH (Gangavati et al., 2011; Harvard Health Publications, 2011). Advanced age and HTN both decrease baroreceptor compensatory mechanisms, which then increase the risk of OH. In both hyper- and hypotension blood vessels may be stiffer than normal rendering baroreceptors less responsive to changes (Harvard Health Publications). According to the literature, people with well-controlled HTN tend to have less occurrences of OH than people with uncontrolled HTN (Gangavati et al.).
What are the signs and symptoms of orthostatic hypotension
OH can be symptomatic or asymptomatic, mild or severe, and acute or chronic (Poon & Braun, 2005). Symptoms can vary greatly from diminished cognition and disturbed emotional status (Mosnaim et al., 2010), to lightheadedness, dizziness and changes in vision (Harvard Health Publications, 2011), to syncope/fainting (Texas Arrhythmia Institute, 2010), and death (Romero-Ortuno et al., 2011). Other common symptoms of OH include weakness, fatigue, nausea, palpitations, and headache. Syncope, dyspnea, chest pain, and neck and shoulder pain are also possible but occur less frequently (Lanier et al., 2011).
Controversy exists over whether or not OH can be linked to falls. Healthcare professionals tend to automatically associate the two; however, Gangavati and colleagues (2011) state that little data exist linking falls with OH based on the current definition of OH. The definition describes a drop in pressure occurring after standing for 3 minutes. By 3 minutes baroreceptors may have begun the compensatory mechanisms; thus, a normal blood pressure may be recorded despite a possible significant drop in pressure earlier than 3 minutes. Findings from Gangavati and associate’s study suggests that if pressure is taken at 1 minute after standing, symptoms may occur, blood pressure may drop, and falls may be the result, thereby possibly linking OH with falls in older adults.
Nursing Diagnosis for Orthostatic Hypotension
Your doctor may review your medical history and your symptoms and conduct a physical examination to help diagnose your condition.
Your doctor also may recommend one or more of the following:
- Blood pressure monitoring. Your doctor will measure your blood pressure both while you’re sitting and while you’re standing and will compare the measurements. Your doctor will diagnose orthostatic hypotension if you have a drop of 20 millimeters of mercury (mm Hg) in your systolic blood pressure or a drop of 10 mm Hg in your diastolic blood pressure within two to five minutes of standing, or if standing causes signs and symptoms.
- Blood tests. These can provide information about your overall health, including low blood sugar (hypoglycemia) or low red blood cell levels (anemia), both of which can cause low blood pressure.
- Electrocardiogram (ECG or EKG). This noninvasive test detects irregularities in your heart rhythm or heart structure and problems with the supply of blood and oxygen to your heart muscle.
During this test, soft, sticky patches (electrodes) are attached to the skin of your chest, arms and legs. The patches detect your heart’s electrical signals while a machine records them on graph paper or displays them on a screen.
Sometimes, heart rhythm abnormalities come and go, and an ECG won’t find problems. If this happens, you may be asked to wear a 24-hour Holter monitor to record your heart’s electrical activity as you go about your daily routine.
- Echocardiogram. In this noninvasive exam, sound waves are used to produce a video image of your heart. Sound waves are directed at your heart from a wandlike device (transducer) that’s held on your chest.
The sound waves that bounce off your heart are reflected through your chest wall and processed electronically to provide video images of your heart in motion to detect structural heart disease.
- Stress test. A stress test is performed while you’re exercising, such as walking on a treadmill. Or you may be given medication to make your heart work harder if you’re unable to exercise. Your heart is then monitored with electrocardiography, echocardiography or other tests.
- Tilt table test. A tilt table test evaluates how your body reacts to changes in position. You’ll lie on a flat table that tilts to raise the upper part of your body, which simulates the movement from lying down to standing. Your blood pressure is taken frequently as the table is tilted.
- Valsalva maneuver. This noninvasive test checks the functioning of your autonomic nervous system by analyzing your heart rate and blood pressure after several cycles of a type of deep breathing: You breathe in deeply and push the air out through your lips, as if you were trying to blow up a stiff balloon.
Nursing Treatments for Orthostatic Hypotension
The goal in treating OH is to improve symptoms without causing supine HTN (Lanier et al., 2011). This includes treating physiological causes, omitting or lowering contributing medications when possible, and using pharmacologic means as a last resort when necessary.
Treatment for acute or chronic OH begins with educating patients about the diagnosis. Following education, nonpharmacologic treatment is the first line of defense. Such interventions include avoiding carbohydrate rich meals, adding sodium-rich foods or sodium tablets to the diet, limiting alcohol intake, and ensuring adequate hydration (Mosnaim et al., 2010).
It has been suggested that elevating the bed 10[degrees]-20[degrees] may decrease the occurrence of OH by slightly decreasing kidney perfusion at night, thus decreasing urine output, and fluid loss. It is recommended that certain medications (especially antihypertensive prescriptions) be taken at bedtime to relieve OH symptoms (Mosnaim et al.; Lanier et al., 2011).
When pharmacologic means are necessary to treat OH, the first line of medications ordered is mineralocorticoids, specifically Fludrocortisone. It works by increasing the absorption of sodium and water and thus raising blood pressure. This drug has several potential adverse effects including headache, supine HTN, hypokalemia, and congestive heart failure.
Another drug used for OH is Midodrine, an a-1-adrenergic agonist. Several potential adverse effects exist for this drug as well. Finally, Pyridostigmine, a cholinesterase inhibitor may be used, but also may have adverse effects including loose stool, diaphoresis, and hypersalivation. Given the potential dangers of the medications, nonpharmacologic treatment of OH is preferred whenever possible (Lanier et al., 2011).
Implications for Home Care and Hospice Nurses: Assessment and Patient Education
Care of the client with OH includes assessing blood pressure while lying, sitting, and standing, and evaluating for orthostatic changes along with symptoms. Careful fall risk assessments, review of high-risk medications, and awareness of contributing diagnoses can alert the nurse to potential risks for OH. Uncontrolled HTN may also be a factor to assess for when considering patients at risk.
Patient education focused on ways to decrease OH is a valuable tool and could potentially save lives. Simple instructions include teaching patients to rise slowly from lying to standing and allowing feet to dangle first while wiggling/flexing toes and feet, which allows blood time to push upward from the periphery (Harvard Health Publications, 2011). Encouraging an exercise program may improve cardiac health, thus helping to avoid OH.
Regimens may also include isometric exercises of the extremities, toe raises, and thigh contractions (Lanier et al., 2011). Such exercise routines may require the input of a physical therapist. Educating patients about dehydration and ways to avoid it is critical. Encouraging small frequent meals to avoid postprandial OH related to large carbohydrate-rich meals may also be helpful (Mosnaim et al., 2010).
Some resources suggest the use of compression stockings to avoid peripheral blood pooling (Mosnaim et al.); however, they may only be helpful if waist high (Harvard Health Publications, 2011), making them somewhat uncomfortable and difficult to apply. Teaching patients to keep a diary of symptoms so healthcare professionals can track patterns may also be helpful (Lanier et al.).
Reviewing medications with patients and teaching them about potential side effects and how to prevent them may be critical in decreasing OH and falls. Finally, patients who have access to telemonitoring systems from their home care agencies can benefit from education focused on the need to check blood pressure sitting, and then 1 and 3 minutes after standing. Information gathered in this way may be useful for titrating medications during physician visits.
Many factors contribute to OH in older adults. Symptoms can range from discomfort to injury and death, while falls may also be associated with OH. Changes in the autonomic nervous system, the heart and vessels, comorbidities, and the use of certain medications may all contribute to OH as people age. Nonpharmacologic methods for treating OH are preferred, and nursing interventions can best focus on patient education. Home care nurses can easily assess for orthostatic changes and can intervene by educating the patients, and alerting the provider about reported and/or observed symptoms.
Nursing Care Plan for Orthostatic Hypotension
Attached below is an example of a nursing care plan for orthostatic hypotension.
Centers for Disease Control and Prevention. (2011). Falls among older adults: An overview. http://www.cdc.gov/HomeandRecreationSafety/Falls/adultfalls.htm
Fortinsky, R. H., Baker, D., Gottschalk, M., King, M., Trella, P., & Tinetti, M. E. (2008). Extent of implementation of evidence-based fall prevention practices for older patients in home health care. Journal of the American Geriatrics Society, 56(4), 737-743.
Gangavati, A., Hajjar, I., Quach, L., Jones, R. N., Kiely, D. K., Gagnon, P., & Lipsitz, L. A. (2011). Hypertension, orthostatic hypotension, and the risk of falls in a community-dwelling elderly population: The maintenance of balance, independent living, intellect, and zest in the elderly of Boston study. Journal of the American Geriatrics Society, 59(3), 383-389.
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Hogan-Quigley, B., Palm, M., & Bickley, L. (2012). Bates’ nursing guide to physical examination and history taking. Philadelphia, PA: Lippincott Williams & Wilkins.
Karch, A. (2006). Focus on nursing pharmacology. (3rd ed.). Philadelphia, PA: Lippincott Williams & Wilkins.
Lanier, J. B., Mote, M. B., & Clay, E. C. (2011). Evaluation and management of orthostatic hypotension. American Family Physician, 84(5), 527-536.
Mosnaim, A. D., Abiola, R., Wolf, M. E., & Perlmuter, L. C. (2010). Etiology and risk factors for developing orthostatic hypotension. American Journal of Therapeutics, 17(1), 86-91.
Poon, I. O., & Braun, U. (2005). High prevalence of orthostatic hypotension and its correlation with potentially causative medications among elderly veterans. Journal of Clinical Pharmacy & Therapeutics, 30(2), 173-178.
Romero-Ortuno, R., Cogan, L., Foran, T., Kenny, R. A., & Fan, C. (2011). Continuous noninvasive orthostatic blood pressure measurements and their relationship with orthostatic intolerance, falls, and frailty in older people. Journal of the American Geriatrics Society, 59(4), 655-665.
Texas Arrhythmia Institute. (2010). Syncope (fainting). Retrieved from http://www.txai.org/edu/syncope/mechanisms.htm.